A síndrome de Legg-Calvé-Perthes ou doença de Perthes é uma doença degenerativa da articulação da anca (quadril em português brasileiro) em que ocorre. El absceso del psoas es una enfermedad infrecuente en niños. Puede tener una en pediatría es primario; sin embargo, en ocasiones, puede ser de origen secundario y médula espinal, enfermedad de. Legg-Calvé-Perthes, entre otros Download Citation on ResearchGate | Enfermedad de Legg Calvé Perthes (LCP) en varios aspectos de la enfermedad de Legg Calvé Perthes y la evolución de caderas de August · Anales espanoles de pediatria.
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The remaining patients were considered withinthe normal range when age was taken into account. Blood, 84pp.
Idiopathic osteonecrosis, hypofibrinolysis, high plasminogen activator inhibitor, high Lp aand therapy with stanozolol. Association of idiopathic venous thromboembolism with single point-mutation at Arg of factor. Alterations in hemostasis and thrombosis were detectedin one patient who had moderate-to-light protein Sdeficiency.
Thromb Haemost, 62pp. J Pediatr Orthop, 19 enfermedax, pp. Perthes’ disease and the relevance of thrombophilia. Conclusions Epidemiological and laboratory data from this group ofpediatric patients and from the control group do not supportthe hypothesis that a thrombogenesis defect couldbe the underlying cause of avascular necrosis of the hipjoint.
Enfermedad de Legg-Calve-Perthes
Clin Chem, 32pp. Resistance to activated protein C and Legg-Perthes disease.
Familial hypofibrinolysis pediaatria venous thrombosis. J Pddiatria Invest, 94pp. Blood, 85pp. It has been suggested that pethes cause enfermedxd Perthes diseasemay be intravascular thrombosis induced by a potentialcongenital hemostatic disorder leading to conditions ofthrombophilia or hyperfibrinolysis. Identification of the same factor V gene mutation in 47 of 50 thrombosis-prone families with inherited resistance to activated protein C.
Mutation in blood coagulation factor V associated with resistance to activated protein C. Association of antithrombotic factor deficiencies and hy-pofibrinolysis with Legg-Perthes disease.
Blood, 87pp. Thromb Haemost, 69pp. Background It has been suggested that the cause of Perthes diseasemay be intravascular thrombosis induced by a potentialcongenital hemostatic disorder leading to conditions ofthrombophilia or hyperfibrinolysis.
Thromb Haemost, 71pp. The Kinston antiphospholipid group. Anal Biochem,pp. Continuing navigation will be considered as acceptance of this use. Am J Hematol, 48pp. Decreased fibrynolitic potential in patients with idio-pathic avascular necrosis and transient osteoporosis of the hip. Am J Clin Pathol, 94pp. J Med,pp.
Am J Hematol, 44pp. High risk of thrombosis in patients homozigous for factor V Leiden activated protein C resistance. Severe thrombotic disease in a young man with bone marrow and skeletal changes and with a high content of an inhibitor in the fibrinolytic system.
Activated protein C resistance as an additional risk factor for thrombosis in protein C deficient families. Maturation of the hemostatic system during childhood.
Clin Orthop,pp. Patients and method Complete study of hemostasis with coagulation and antithrombinfactors as well as study of thrombophilia andfibrinolysis in these patients could determine the prevalenceof hemostasis and fibrinolysis in this group of patients. Clin Othop,pp. N Engl J Med,pp.
¿Trombofilia y enfermedad de Perthes? | Anales de Pediatría
Blood, 82pp. Am J Hematol, 45pp. Epidemiological and laboratory data from this group ofpediatric patients and from the control group do not supportthe hypothesis that a thrombogenesis defect couldbe the underlying cause of avascular necrosis of the hipjoint.
The second international anticardiolipin standardization workshop. Blood collection in strongly acid citrate anticoagulant used in a study of dietary influence of basal Tpa activity. To improve our services and products, we use “cookies” own or third parties authorized to show advertising related to client preferences through the analyses of navigation customer behavior. Mutation in gene coding for factor V and the risk of myocardial infarction, stroke and venous thrombosis in apparently healthy men.
pertbes Lancet,pp. Genetics aspects of Perthes disease: J Lab Clin Med,pp. Aguirre Canyadell aI. The remaining patients were considered withinthe normal range when age was taken into account.
Non traumatic osteonecrosis of the femoral head: You can change the settings or obtain more information by clicking here.