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The data indicated that GBE repressed glucose uptake under normal conditions, while it dramatically improved glucose tolerance under insulin-resistant conditions. Abstract Our aim is to investigate the potential therapeutic value of morin against leei and elucidate the mechanism of action.
Gene Ontology GO Terms.
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It is said that GBE treatment could avoid drug-induced obesity. To better show its efficacy, we included a control group that was treated with rosiglitazone, a type of thiazolidinedione TZD. How does Europe PMC derive its citations network? Find all citations in this journal default.
Journal of Natural Medicines [24 Jul72 4: Morin treatment also increased the number of trabecular bones in DEX-induced rats. Pathological examination was performed by hematoxylin and eosin staining. Read Article at publisher’s site. Our data suggest that GBE has the potential to prevent insulin ,ei and is a promising anti-diabetic drug.
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Here, we uncovered the therapeutic effect of morin against osteoporosis and demonstrated its suppressive action on the MAPK pathway in this disease. Journal of Natural Medicines [03 Sep65 1: The relative expression of osteogenic and bone resorption markers was determined by real-time polymerase chain reaction and Western blotting, respectively. Body mineral density BMD was determined at proximal femurs using dual energy X-ray absorptiometry.
Gene Ontology GO Terms. Mechanistically, morin reversed the decrease of osteogenic markers and increase of bone resorption markers, which might eventually be mediated by modulation of MAPK signaling cascades.
No matching affiliation detected. CitePeer Related Articles http: In the current study, we tested the hypothesis that Ginkgo biloba extract GBE prevented hyperinsulinism-induced glucose intolerance in hepatocytes.
Furthermore, after analyzing gene expression, we suggest that GBE chiefly exerts its effects by stimulating IRS-2 transcription.
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